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BiP Up-Regulation in JEV-Infected BHK-21 Cells by jkz8zpbpvb is a document available to read on EtoBox.

This study investigates the mechanisms of persistent Japanese Encephalitis Virus (JEV) infection in BHK-21 cells, focusing on the role of the unfolded protein response (UPR) factors. The researchers found that constant up-regulation of BiP expression prevents virus-induced apoptosis by completely depleting CHOP levels, allowing the cells to survive despite active viral replication. These findings suggest that the modulation of UPR pathways is crucial for the persistence of JEV in mammalian cells.

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jkz8zpbpvb
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