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This study investigates the effects of platelet-rich plasma (PRP) on tendon fibroblasts and macrophages, revealing that PRP activates proinflammatory signaling pathways, particularly TNFa and NFkB, and induces oxidative stress in fibroblasts. The research shows that PRP downregulates extracellular matrix gene expression and does not significantly affect macrophage polarization. These findings suggest that PRP may initiate a transient inflammatory response that could promote tissue regeneration in tendon inj

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xmb8jf6vch

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EN