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This review discusses the potential of FcRn inhibitors as a novel treatment for myasthenia gravis, an autoimmune disease characterized by impaired neuromuscular transmission due to autoantibodies. By antagonizing the neonatal Fc receptor (FcRn), these inhibitors can accelerate the clearance of pathogenic immunoglobulin G (IgG), offering a promising alternative to current therapies. The review highlights recent research advancements and the approval of efgartigimod by the FDA for generalized myasthenia gravi

Author

Sandra Iglesias Perez

Language

EN