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1) Arterial wall injury, such as endothelial injury, allows lipids and platelets to accumulate in the area, increasing permeability. 2) Oxidized LDL attracts monocytes and macrophages, which begin to form plaques as lipids are engulfed by foam cells. 3) Smooth muscle cells multiply within the plaques, decreasing coronary tissue perfusion and oxygenation, which can lead to angina or myocardial infarction.

Author

shaitabligan

Language

EN