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This study investigates the role of bystander activated CD8+ T cells in mediating neuropathology during Zika virus (ZIKV) infection, revealing that neurological disease severity correlates with CD8+ T cell infiltration rather than viral load. The research demonstrates that these T cells exhibit antigen-independent cytotoxicity, primarily through NKG2D expression, contributing to brain damage. The findings suggest potential therapeutic targets for treating virus-induced neurological disorders by modulating C

Author

tzuy0707

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EN