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This study investigates the dysfunction of estrogen signaling in granulosa cells of women with polycystic ovary syndrome (PCOS), revealing significant alterations in gene expression and steroid concentrations. The findings indicate that estradiol (E2) signaling is ineffective in regulating key endocrine genes in PCOS, contributing to impaired folliculogenesis. Overall, the research highlights the molecular signatures associated with PCOS, particularly in non-hyperandrogenic overweight women.
- Author
- bou
- Language
- EN