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Sepsis causes relative hypovolemia due to decreased vascular resistance, increased venous capacitance, and vascular leak, which decreases stroke volume and cardiac output, resulting in tissue hypoxia, anaerobic metabolism, and lactic acidosis. The targets of fluid resuscitation for sepsis are to maintain central venous pressure between 8-12 mmHg with 500mL crystalloids every 30 minutes, mean arterial pressure at 65 mmHg using vasopressors and vasodilators, SvO2 at least 70% through blood transfusion or dobu

Author

Annisa Safira Nurdila

Language

EN