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The document discusses the tumor microenvironment (TME) in pancreatic ductal adenocarcinoma (PDAC), highlighting its cellular and non-cellular components that contribute to tumor progression and therapy resistance. Key elements include cancer-associated fibroblasts (CAFs), immune cells, and the extracellular matrix (ECM), which interact to promote tumor growth and suppress immune responses. The document emphasizes the potential for targeting these components to improve treatment efficacy and overcome therap

Author

rumeysaadiyil

Language

EN