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The study investigates the role of Epstein-Barr virus (EBV) in systemic lupus erythematosus (SLE), revealing that EBV infection reprograms autoreactive B cells into antigen-presenting cells, thereby promoting autoimmunity. Using a novel EBV-specific single-cell RNA sequencing platform, researchers found increased frequencies of EBV+ memory B cells in SLE patients, which express genes associated with immune activation and antigen presentation. The findings provide mechanistic insights into how EBV may drive

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asd

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