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Chloramphenicol inhibits bacterial protein synthesis by binding to the 50S ribosomal subunit, preventing peptide bond formation. Resistance to the drug can occur through enzyme production, decreased cell wall permeability, or ribosomal mutation. It is primarily bacteriostatic but can be cidal at high concentrations, and may cause gastrointestinal effects and gray baby syndrome in neonates.

Author

Sejal Khuman

Language

EN