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The article reviews vascular changes associated with bleomycin-induced scleroderma, a mouse model for systemic sclerosis (SSc). It highlights the role of vascular injury and dysfunction in the pathogenesis of SSc, emphasizing the interactions among various cell types and the impact of cytokines and reactive oxygen species. The findings suggest that targeting vascular changes may offer therapeutic benefits in managing scleroderma-related fibrosis.

Author

Sally Eshiba

Language

EN