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Diabetes mellitus, particularly type 1 diabetes (T1D), results from a loss or dysfunction of insulin-producing pancreatic β-cells. Restoration of β-cell mass and function remains a major therapeutic goal. Two complementary strategies hold promise: endogenous β-cell regeneration, through stimulating proliferation, neogenesis, or transdifferentiation, and exogenous replacement using stem-cell-derived islet (SC-islet) transplantation. This review synthesizes recent advances in both areas, delin

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EN