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This thesis investigates the role of Rho GEF Arhgef2 in RAS-mediated tumorigenesis, highlighting its upregulation in RAS-mutated tumors and its essential function in promoting cell survival and tumor growth. Arhgef2 acts as a positive regulator of the MAPK pathway, contributing to the malignant characteristics of tumor cells and their potential for metastasis. The findings suggest that targeting Arhgef2 may offer a new therapeutic strategy for treating RAS-mutated cancers.

Author

jiaoying1994

Language

EN