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The study investigates the role of leptin receptor-expressing neurons in the nucleus tractus solitarius (NTS) in suppressing food intake independently of GLP1 in mice. It finds that while GLP1NTS neurons can reduce food intake, LepRbNTS neurons are more effective and durable in their suppression of appetite, indicating that leptin signaling plays a dominant role. Ablation of Ppg in the NTS does not significantly affect energy balance, suggesting that other pathways are crucial for long-term control of food

Author

Zhaoyue Zhang

Language

EN