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What is Srep 20321 about?

The study investigates the quorum sensing inhibitor Hamamelitannin (HAM) and its ability to increase the antibiotic susceptibility of Staphylococcus aureus biofilms. It demonstrates that HAM affects biofilm susceptibility by altering peptidoglycan biosynthesis and extracellular DNA release, enhancing the effectiveness of various antibiotics in vitro and in vivo. The findings suggest that targeting quorum sensing could be a promising strategy to combat antibiotic resistance in S. aureus infections.

Author

Rahim H

Language

EN