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This review discusses the dual role of reactive oxygen species (ROS) in the mammalian central nervous system, highlighting their potential to both damage cellular components and facilitate synaptic plasticity. While excessive ROS accumulation is linked to cognitive decline and neurodegenerative disorders, controlled ROS production is essential for activating signaling pathways involved in synaptic changes. The document explores the sources of ROS in neurons and their involvement in both physiological and pa

Author

fdctpftftr

Language

EN