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This study investigates the impact of HLA-mediated immune pressure on the replication capacity of HIV-1 subtype C in chronic infection. It finds that protective HLA-B alleles, particularly HLA-B*81, are associated with lower viral replication capacities and that specific mutations in Gag-protease can influence viral fitness. The findings suggest that immune-driven sequence changes in Gag may play a role in HIV-1 pathogenesis, although the long-term clinical benefits of these changes remain uncertain.

Author

rohin athavale

Language

EN