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This document summarizes cytoadherence in malaria infections. Cytoadherence occurs when red blood cells infected with Plasmodium falciparum bind to endothelial cells in blood vessels. This binding causes sequestration and rosetting, which obstruct blood flow and damage capillary walls. Several human receptors, such as CD36, ICAM-1, and ELAM-1, bind to parasite ligands like PfEMP-1, contributing to severe malaria pathogenesis. Intervention in the cytoadherence process through blocking specific ligands or the

Author

Syarofis Si'ayah

Language

EN