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This study investigates the cellular and molecular mechanisms of diabetic retinopathy (DR) using human retinal organoids to model incremental hyperglycaemic stress. The findings reveal that early-stage organoids are resilient to modest glucose increases, while late-stage organoids show significant vulnerability, including photoreceptor dysfunction and increased apoptosis. This research highlights the potential of human retinal organoids as a platform for understanding neurodegeneration in DR and identifying

Author

dan.torresug20

Language

EN