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This study investigates the role of dendritic cells and anergic type I NKT cells in sulfatide-mediated immune regulation during experimental autoimmune encephalomyelitis (EAE). Activation of sulfatide-reactive type II NKT cells significantly reduces pathogenic T cell activity and promotes immune tolerance, suggesting a novel therapeutic pathway for autoimmune diseases. The findings indicate that targeting the sulfatide-mediated immune regulatory pathway could lead to non-HLA-dependent treatments for T cell-

Author

Muhammad Athamna

Language

EN