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Caspase activation is generally thought to be necessary to induce apoptosis. This study tests the hypothesis that caspase-independent apoptotic pathways contribute to the development of heart failure in the setting of caspase inhibition. Early (6 day) survival was significantly better in CrmA Tg (81%) than WT mice (38%). Twelve days after DOX injection, the mortality benefit had dissipated.

Author

Rafael Dall'Alba

Language

EN