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This review discusses the critical role of dopamine D2 receptors (D2Rs) in compulsive eating and obesity, highlighting how chronic high-fat food consumption leads to D2R downregulation and desensitization. This impairment disrupts downstream signaling pathways, particularly the cAMP/PKA/pCREB pathway, which alters the expression of neuropeptides that regulate appetite and food reward. The findings suggest that these changes contribute to binge-eating behaviors and a cycle of reward hypofunctionality, exacer

Author

reer

Language

EN