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The study investigates the role of myogenin, a muscle-specific transcription factor, in shifting enzyme activity from glycolytic to oxidative metabolism in transgenic mice. Overexpression of myogenin led to increased levels of oxidative mitochondrial enzymes and decreased levels of glycolytic enzymes, resulting in a metabolic shift without altering myosin heavy chain isoform expression. These findings suggest that myogenin may mediate the effects of physical training on muscle metabolic capacity.
- Author
- Manuel Hernández
- Language
- EN