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What is Death Receptor-Induced Apoptotic and Nonapoptotic Signaling about?

## Glossary Apoptosis Apoptosis is a form of cell death morphologically characterized by cell shrinkage, chromatin condensation and fragmentation, and plasma membrane blebbing. Even though it is not immunologically silent, apoptosis is usually considered as anti-inflammatory, contrary to necrosis. Apoptosis requires the activation of caspases. It can be exogenously induced by binding of death ligands to their receptors and endogenously by cellular stressors, for example, DNA damage. Caspases Caspases (for cysteinyl-dependent aspartatespecific proteases) are enzymes that belong to a family of endopeptidases that cleave their substrates after an aspartic acid residue and possess a cysteine within their catalytic site. Complex II Secondary cytoplasmic signaling platform composed of a number of factors that also form part of complex I (respectively, the DISC for CD95L and TRAIL or the TNF-RSC for TNF and TL1A) including the adaptor molecules FADD or TRADD and other molecules recruited from the cytoplasm. This complex II will trigger the secondary function of each receptor, that is, for the CD95L and TRAIL systems gene induction and proinflammatory signaling and for the TNF system induc

Author

C. Kantari; E. Lafont; H. Walczak

Publisher

Elsevier Science & Technology Books

Published

2014

Language

EN

ISBN

9780123864574

Updated

2026-03-24