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The study investigates the role of the kynurenine pathway in endothelial cell apoptosis and dysfunction induced by angiotensin II (AngII) in mice. It reveals that AngII activates NAD(P)H oxidase, leading to increased reactive oxygen species and endothelial cell death, which is dependent on interferon-gamma (IFN-γ) and the production of tryptophan-derived metabolites. The findings suggest that targeting this pathway may provide therapeutic strategies for cardiovascular diseases associated with endothelial dy

Author

thanushreeva

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EN