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This study examined the role of nitric oxide in the development of haloperidol-induced supersensitivity. Rats were pretreated with either saline or the nitric oxide inhibitor L-NAME during chronic haloperidol treatment. Rats pretreated with L-NAME showed significantly less cocaine-stimulated locomotion after haloperidol treatment than rats pretreated with saline, suggesting that nitric oxide is involved in haloperidol-induced supersensitivity. Inhibiting nitric oxide synthesis may decrease side effects of l

Author

Lonkes

Language

EN