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This study investigates how cochlear amplification affects synaptic transmission at the endbulb of Held synapse in the cochlear nucleus, using Prestin-/- mice to eliminate cochlear amplification. The findings reveal that the absence of cochlear amplification leads to increased excitability of bushy cells and significant alterations in synaptic transmission, including reduced synaptic strength and a reversal of paired-pulse plasticity. This research highlights the interplay between peripheral cochlear mechan

Author

Maria Eugenia Gomez-Casati

Language

EN