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Prostaglandins Modulate the Responsiveness of the Gastric Microcirculation of Sodium Nitroprusside in Cirrhotic Rats by J Geraldo; P Ferraz; J L Wallace is a Medicine article available to read on EtoBox.

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Portal hypertensive gastropathy is considered a serinol-induced gastric injury, related to an inability to ous complication observed in patients with cirrhosis mount a defensive gastric hyperemic response to lumiand portal hypertension. 1,2 The pathophysiology of the nal irritants, and associated with an impaired reactivity profound alterations in mucosal defense against injury of the gastric microcirculation to nitric oxide (NO)/cyclic observed in portal hypertension have demonstrated guanosine monophosphate (cGMP)-dependent vasodilathat the increased susceptibility of the gastric mucosa tors. Whether this hyporesponsiveness is in some way to injury was intimately related to vascular abnormalirelated to depressed prostaglandin synthesis by the ties in the gastric microcirculation. [3][4][5] stomach of cirrhotic rats is not clear. The aims of this The gastric hyperemic response to irritants is a critistudy were to evaluate the role of NO and prostaglandins cal component of mucosal defense. It is responsible for in the regulation of the gastric microcirculation under resting conditions and in response to administration of the buffering of back-diffused acid and dilution and sodium n

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Author

J Geraldo; P Ferraz; J L Wallace

Publisher

John Wiley and Sons; Wiley (John Wiley & Sons); John Wiley & Sons Inc.; Elsevier BV; Wiley; Research Square (ISSN 0270-9139)

Published

1996

Language

EN

Field

Medicine (Health Sciences)