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Resolution of post-lung transplant ischemia-reperfusion injury is modulated via Resolvin D1-FPR2 and Maresin 1-LGR6 signaling 1 Materials and methods 2 Lung IR injury model 2 Human BAL analysis 2 Deceased cardiac donor (DCD) and orthotopic lung transplantation 2 Statistical analysis 2 Results 2 Endogenous increase in SPM expression occurs after lung transplantation 2 Early pulmonary dysfunction after IR injury is attenuated by RvD1 and/or MaR1 treatment 3 Lung inflammation and injury after IR are effectively attenuated by concomitant treatment with RvD1 and MaR1 4 RvD1 decreases alveolar macrophage activation via FPR2 receptors and MaR1 decreases ATII epithelial cell activation via LGR6 receptors 5 Efferocytosis of neutrophils is increased by RvD1/FPR2 signaling 6 Post-transplant lung injury is significantly ameliorated by RvD1 and MaR1 treatment in DCD donors 7 Discussion 7 Author contributions 12 Disclosure statement 12 Supplementary materials 12 References 12

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UND

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nonfiction