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Mechanisms of term and preterm birth involve activation and stimulation of the myometrium. Activation can occur through mechanical stretching of the uterus or endocrine pathways resulting from increased activity of the fetal hypothalamic-pituitary-adrenal axis. Cortisol likely contributes to increased prostaglandin production in fetal tissues through up-regulation of prostaglandin H synthase-2 and down-regulation of 11β-hydroxysteroid dehydrogenase. Oxytocin, produced locally within the intrauterine tissues

Author

Chenso

Language

EN