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Cerebral salt wasting syndrome is characterized by hyponatremia with elevated urine sodium and hypovolemia that can occur after central nervous system insults like subarachnoid hemorrhage. It is debated whether it is a distinct condition or a form of SIADH. Treatment involves volume repletion with isotonic or hypertonic saline along with sodium supplementation to restore sodium stores, as opposed to fluid restriction for SIADH. Distinguishing the two conditions is important as they have opposing treatment a

Author

Onggo Wiliyanto

Language

EN