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This review discusses the critical role of thyroid hormone (TH) in skeletal development and maintenance, highlighting its effects on various bone cell types and the mechanisms of action through nuclear receptors. It emphasizes the importance of TH in regulating growth factor signaling pathways that influence bone metabolism, as well as the consequences of TH dysregulation in both genetic mouse models and clinical cases. The review aims to clarify the molecular mechanisms underlying TH

Author
kkkkkkkkkkkkkk
Language
EN

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