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This research article investigates the effects of polyphyllin I (PPI) on the secretion of IL-8 in HaCaT cells induced by Propionibacterium acnes (P. acnes) through the CD36/NOX1/ROS/NLRP3/IL-1β pathway. The study finds that PPI inhibits the activation of this pathway, thereby reducing IL-8 secretion and keratinocyte proliferation and migration, suggesting its potential as a novel therapy for acne vulgaris. The findings contribute to understanding the molecular mechanisms of acne pathogenesis and highlight P

Author

henrysnight

Language

EN