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The biological basis of keloid formation involves substantial proliferation and apoptosis inhibition of fibroblasts, as well as an imbalance between collagen synthesis and degradation. Keloid fibroblasts proliferate more rapidly and overproduce type I collagen fibers while highly expressing growth factors like TGF-β1 and PDGF-α. Intralesional 5-FU is used to treat keloids by inhibiting fibroblast proliferation and interrupting DNA/RNA synthesis as well as inhibiting TGF-β-induced collagen production. A stud
- Author
- Andriantk
- Language
- EN