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This study investigates the effects of methylglyoxal-modified albumin (MGA) on arginase activity and vascular function in endothelial cells, highlighting the role of advanced glycation end products (AGEs) in vascular dysfunction associated with diabetes. The findings indicate that MGA increases arginase activity and expression, impairing nitric oxide production and vasorelaxation, effects that can be reversed by arginase inhibition. This research suggests that AGEs may contribute to vascular dysfunction in

Author

Ebaa Moh'd Zayadneh

Language

EN