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This study investigates the role of the aryl hydrocarbon receptor (AHR) in the adaptive reprogramming of small intestinal epithelial cells, highlighting its impact on cellular proliferation, differentiation, and intestinal resilience. Genetic ablation of Ahr impairs the differentiation of secretory cell lineages and decreases intestinal barrier function, while AHR activation through dietary ligands, such as those from broccoli, enhances gene expression related to goblet cell differentiation. The findings su

Author

kxtmc4bpq6

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EN