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Cutaneous lupus erythematosus (CLE) is an autoimmune disease that can present as isolated skin lesions or as a manifestation of systemic lupus erythematosus. Skin lesions in CLE are characterized by interface dermatitis, an anti-epithelial inflammatory reaction at the dermo-epidermal junction orchestrated by type I and III interferon-regulated cytokines and chemokines. Both innate and adaptive immune pathways are strongly activated in CLE lesions due to continuous reactivation of innate pathways via pattern

Author

Marlin Berliannanda Taway

Language

EN