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Emodin alleviates cardiac fibrosis by suppressing activation of cardiac fibroblasts via upregulating metastasis associated protein 3 (MTA3). The study examined the effects of emodin on cardiac fibrosis induced by transaortic constriction or angiotensin II in mice. Emodin administration decreased left ventricular wall thickness and suppressed cardiac fibroblast activation. Emodin upregulated expression of MTA3 and restored MTA3 expression in cardiac fibrosis. Overexpression of MTA3 promoted fibrosis, while s

Author

Mohammed Shuaib Ahmed

Language

EN