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This study investigates the cognitive impairments in mice lacking adenosine A2A receptors (A2AR) and their association with changes in synaptic protein expression and neurogenesis. The results indicate that A2AR knockout mice exhibit significant cognitive deficits linked to decreased hippocampal neuron proliferation and alterations in synaptic proteins. These findings suggest that impaired adenosine signaling may contribute to cognitive deficits in psychiatric disorders, highlighting potential therapeutic a

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ensargalataaslan

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EN