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This study aimed to identify biomarkers that could differentiate between sepsis-associated encephalopathy (SAE), sepsis without brain dysfunction, and delirium from other causes using a sample of 30 patients with community-acquired pneumonia-induced sepsis, of which 10 developed SAE. They measured levels of biomarkers related to inflammation, endothelial activation, coagulation, and brain function. They found that only brain-derived neurotrophic factor (BDNF), regulated upon activation normal T cell express

Author

Cristiane Tomasi

Language

EN