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This study investigates the mechanism of minoxidil in treating androgenetic alopecia (AGA) and identifies two new targets, CYP17A1 and CYP19A1, alongside androgenic receptors. Through network pharmacology and experimental validation, it was found that minoxidil alters hormonal and enzymatic pathways, inhibiting AR and affecting dihydrotestosterone and estradiol levels. The findings suggest new therapeutic targets for AGA treatment and potential avenues for drug development.

Author

Karen Soto

Language

EN