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This study investigates the effects of Cucurbitacin B (CuB) on rheumatoid arthritis (RA) using a collagen-induced arthritis mouse model. CuB significantly reduced arthritis severity and pro-inflammatory cytokine expression by inhibiting Th17 cell differentiation through the JAK/STAT pathway. The findings suggest that CuB may be a promising therapeutic agent for RA by targeting key inflammatory pathways.

Author

eriel

Language

EN