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The document presents a novel hypothesis that endocrine-disrupting chemicals (EDCs) induce gestational diabetes mellitus (GDM) by epigenetically reprogramming placental trophoblast cells, leading to impaired glucose transport and increased inflammation. It discusses the mechanisms by which EDCs alter DNA methylation and histone modifications, affecting glucose transporter genes and inflammatory pathways. If validated, this hypothesis could inform early detection, prevention strategies, and therapeutic inter

Author

nikteshg

Language

EN