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The study investigates the impact of Cryptosporidium parvum IId subtype on the intestinal epithelial barrier in neonatal mice, revealing that it causes significant damage and increased permeability through the downregulation of cell junction molecules. The research highlights that the IId subtype leads to a more severe infection compared to the IIa subtype, with notable reductions in E-cadherin expression. These findings contribute to understanding the pathogenesis of cryptosporidiosis, particularly in vuln

Author

docisraafahmi

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EN