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This study investigates the mechanisms leading to insulin resistance in skeletal muscle following oral and intravenous lipid loading compared to low-dose endotoxin infusion. The findings indicate that both oral and intravenous fat significantly reduce insulin sensitivity through activation of protein kinase C and accumulation of lipid metabolites, while endotoxin primarily triggers inflammatory pathways. The research highlights the complexity of lipid-induced insulin resistance and the role of innate immune
- Author
- Jothi Prem John
- Language
- EN