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This research investigates the role of hexokinase II (HKII) in promoting collateral sensitivity in multidrug resistant chronic myeloid leukemia (CML) cells. The study finds that detachment of HKII from mitochondria leads to decreased bioenergetic parameters and increased drug accumulation, suggesting a potential therapeutic strategy for overcoming drug resistance in CML. The findings highlight the importance of targeting metabolic pathways to enhance treatment efficacy in chemotherapy-refractory patients.

Author

priya

Language

EN