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Mechanisms of DNA Damage Recognition in Mammalian Cells by Hanspeter Naegeli D.V.M. (auth.) is a nonfiction available to read on EtoBox.

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Freckle-like pigmentation abnormalities Ichthyosis Brittle hair with low sulphur Skin cancer Mental retardation Brain pathology Sexual development Dwarfism Unscheduled DNA synthesis Xeroderma pigmentosum Yes Yes No No Yes Some patients Atrophy Immature in some patients No Reduced (except in the variant form) between nucleotide excision repair and transcription suggests that at least some clinical features of trichothiodystrophy are the result of a transcription defect rather than the consequence of a DNA excision repair insufficiency. For example, the characteristic skin, nail and hair abnormalities observed in trichothiodystrophy patients may relate to defective transcription of genes encoding sulfur proteins of epidermal tissues. Similarly, neurodysmyelination may involve reduced transcription of genes encoding faetors that are important for myelin sheet formation. This possible association between defective transcription and various phenotypic characteristics of trichothiodystrophy led to the suggestion that this disease defines, in part, a "transcription syndrome".8,?O T richothiodystrophy Cockayne syndrome Yes Yes No No Yes No Yes No No No Yes Yes Dysmyelination Dysmyelination

Who reads Mechanisms of DNA Damage Recognition in Mammalian Cells?

Author

Hanspeter Naegeli D.V.M. (auth.)

Publisher

Springer Science & Business Media

Published

1997

Language

EN

ISBN

9780412133114

Category

nonfiction

Subjects

Social Science, Science, Chemistry