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This study investigates the protective effects of aliskiren on atrial structural remodeling induced by rapid atrial pacing in a canine model. Results indicate that aliskiren significantly reduces myocardial fibrosis and the expression of TGF-β1, MEK, and ERK1/2, suggesting its potential as a therapeutic agent for atrial fibrillation. The findings highlight aliskiren

Author

Felipp Silveira

Language

EN