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A gain of function mutation in the ras proto-oncogene can cause cancer by resulting in hyperactive Ras protein that triggers excessive cell division even without growth factors. A loss of function mutation in the p53 tumor suppressor gene can also cause cancer because mutations in both alleles prevent expression of genes that suppress the cell cycle, repair DNA, and induce apoptosis, leading to uncontrolled cell growth.

Author

Chwa Yi Jie

Language

EN