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This study investigates the mechanisms by which pituitary adenylate cyclase activating polypeptide (PACAP) stimulates secretion in chromaffin cells, highlighting the role of Ca2+ influx and mobilization. It demonstrates that PACAP induces sustained increases in cytosolic Ca2+ through both L-type channel influx and inositol trisphosphate receptor-mediated Ca2+ release from the endoplasmic reticulum, with a critical dependence on phospholipase C epsilon (PLCε) activity. The findings enhance understanding of P
- Author
- ds.deepshikha172
- Language
- EN